In 1967 Professor Enno Mandema hosted the first International Symposium on Amyloidosis in Groningen. We are proud to announce that, almost 45 years later, Groningen will be again the venue of the XIIIth Symposium.
This symposium will focus on basic and translational research in the systemic amyloidoses. The systemic amyloidoses are rare diseases caused by clonal immunoglobulin light chains, mutant and wild type transthyretin proteins, serum amyloid A protein, and β2microglobulin that cause disease in all organ systems but most frequently in kidney, heart, gastrointestinal tract and nervous system. The systemic amyloidoses also include diseases caused by a number of inherited mutant proteins, that lead to late onset amyloid fibril deposition in renal, gastrointestinal, and neurologic systems, including the proteins: apolipoprotein AI, apolipoprotein AII, fibrinogen, lysozyme, and gelsolin.
The scientific program will include oral presentations and posters on the process of fibrillogenesis, mechanisms of cell damage and tissue toxicity, emerging diagnostics, and innovative therapeutics. These are current, cutting edge research topics in the systemic amyloidoses.
This symposium will encourage a training environment for junior investigators and trainees. Their presentations will be encouraged. All sessions will provide ample time for discussion after each presentation. We hope to bring together senior investigators, junior investigators, and trainees of all disciplines of science and medicine from a large world-wide community of amyloid researchers.
On behalf of the organizing committee,
Ingrid van Gameren
Groningen Unit for Amyloidosis Research & Development (GUARD)